PMM 3 – Neurological characteristics of the PMM

This is a presentation of a few PMM’s neurological characteristics. They are essential to the understanding of how it functions and will allow you to get a better grip on how important such a system is. They guide us and validate our choice of the neuromuscular spindle and the Golgi organs as the captors of one of the most sensitive functional entity. To highlight the transmission modes of data linked to the PMM, we will use the following tools :

1. North and south poles of magnets which increase or decrease the PMM’s frequency (depending on the tested zone) in a healthy subject.
2. The "Ataëv" method, used by the author of the same name, demonstrated the influence of the subject’s will on muscle activity. It showed that when a tetraplegic was asked to contract a muscle he or she no longer had control of, the temperature of the muscle went up.
3. Full-lungs and empty-lungs retentions in a healthy subject will progressively freeze the PMM’s frequencies.

You will find below a concise comparative chart of the effect of various lesions on the PMM’s frequencies.

We deal with retentions globally (full and empty-lungs) because, although they have different effects on the muscle groups, they indicate the same variations in rhythm.

The muscles tested with magnets are the main fléchisseurs of the tested limbs (brachial biceps or quadriceps). Values are inverted in the antagonist muscles.

The Ataëv techniques can be used for all muscles in the body.

PMM Frequency Apnea Magnets Ataëv south north Healthy subject Normal → 0 ↑ ↓ ↑ medullaire Lesion Normal → 0 ↑ ↓ ↑ Severed nerve Normal → 0 ↑ ↓ =

It is observed that :

1. Willing a muscle to contract influences the PMM in the case of a damaged spinal chord but not in the case of a severed nerve. Information is indeed transmitted through nerves but it can shunt one or several vertebral levels. This phenomenon can be explained either by motivity being carried out by several vertebral levels, or by the use of the ascending and descending pathways of the 1a fibers in the neuromuscular spindles thus crossing over the medullary lesion.
2. During apnea, the PMM stops even in case of a severed nerve, which looks like a mechanical transmission mode through stretching and relaxing of the muscles. Like the heart muscle, the PMM’s activity is semi autonomous, adjusted by the subcortical region (subthalamic nucleus?).
3. Local activity is influenced, even in the case of a nerve completely severed, by a magnet’s north and south poles. So there is a possibility to locally influence the frequencies of muscle contractions without using the nerves (local control).

It can therefore be inferred that the PMM is semi-autonomous, adaptable and controlled by superior nervous centers. Also it seeks to synchronize and adapt itself locally to the general and local circumstances.